ONK Therapeutics

Onk Therapeutics

Biotechnology, Galway Ie, Galway, New York, United States, 11-50 Employees

onktherapeutics.com

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phone no Phone Number: 35**********

Who is ONK THERAPEUTICS

ONK Therapeutics, is an innovative cell therapy company dedicated to developing a next generation of off-the-shelf, dual-targeted NK cell therapy platform targeting hematological malignan...

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  • Galway Ie, Galway, New York, United States Headquarters: Galway Ie, Galway, New York, United States
  • 2015 Date Founded: 2015
  • 11-50 Employees: 11-50
  • dollar-icon Revenue: Under $1 Million
  • tech-icon Active Tech Stack: See technologies

industries-icon Industry: Biotechnology

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Frequently Asked Questions Regarding ONK Therapeutics

Answer: ONK Therapeutics's headquarters are located at Galway Ie, Galway, New York, United States

Answer: ONK Therapeutics's phone number is 35**********

Answer: ONK Therapeutics's official website is https://onktherapeutics.com

Answer: ONK Therapeutics's revenue is Under $1 Million

Answer: ONK Therapeutics has 11-50 employees

Answer: ONK Therapeutics is in Biotechnology

Answer: ONK Therapeutics contact info: Phone number: 35********** Website: https://onktherapeutics.com

Answer: ONK Therapeutics, is an innovative cell therapy company dedicated to developing a next generation of off-the-shelf, dual-targeted NK cell therapy platform targeting hematological malignancies and solid tumors. The Company was founded in 2015, by Prof. ODwyer MD, of NUI Galway, an expert in translational multiple myeloma research, the tumour microenvironment, and exploitation of NK cells as cellular immunotherapy. Its core proprietary platform is based on a dual-targeted NK cell expressing both a chimeric antigen receptor (CAR) targeting a known tumour antigen and a TNF related apoptosis inducing ligand variant (TRAILv) targeting the death receptor pathway (i.e. DR4 or DR5). This promising new approach has the potential to enhance efficacy by addressing both intrinsic (e.g. CAR engagement of a tumor specific antigen) and extrinsic (e.g. signalling through the death receptor pathway) apoptotic pathways, and to reduce the susceptibility to possible target antigen escape through the engagement of tumor antigen independent TRAILv.

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